Tren- great medication, which gives excellent results on the path of "solo" to increase muscle mass without the need to be combined with other medications. However, it is a very powerful steroid, and therefore do not exceed the recommended dosage of certain: for acetate - is 50mg per day for enthatate: 300-350mg per week. To test the tolerance of Tren is better to start with a minimum dosage. If the entire rate is not more than six weeks, the need for additional formulations appears; when 6 to 8 weeks, with the need to enter the second week Gonadotropin (500 / 1000ME every 7 days) and stop taking it two weeks after administration cycle. Next, we have to be post-cycle therapy: start 14 days after the last injection or after 3days (if used Tren Acetate). To restore testosterone production take testosterone boosters (4 weeks after the course).
Given the nature of many of the androgenic and estrogenic side effects from Trenbolone, it is often difficult to minimize the risk of negative effects while still preserving the efficacy of the drug. There are certainly methods that bodybuilders use to minimize the likelihood of harmful reactions, and the most important would be only taking anabolic-androgenic steroids under the supervision of a doctor who is well-versed in steroid use.
I think that if most bodybuilders objectively consider the long-term risks versus the short-term gains, it really won’t seem worth it in the end.
Trenbolone acetate is a modified form of nandrolone.  The structure of trenbolone acetate is a 19-nor classification, which represents a structural change of the testosterone hormone. Trenbolone acetate lacks a carbon atom at the 19 position and carries a double bond at carbons 9 and 11. The position of these carbons slows its metabolism, which greatly increases its binding affinity to the AR, and inhibits it from undergoing aromatization into the corresponding estrogenic metabolite. Trenbolone acetate contains trenbolone modified with the addition of a carboxylic acid ester ( acetic acid ) at the 17β-hydroxyl group.  This facilitates the slow release of the AAS from the area of injection.